Viral hepatitis isn’t quite the scourge it used to be. Thanks in large part to widespread immunization of adolescents and young children in the U.S. for hepatitis A and B, the incidence of the liver-destroying disease has fallen 90% in the past 20 years. Yet many people who could be vaccinated against hepatitis haven’t been -- and so remain at higher risk.
Hepatitis C is a sneaky virus. You may not have any symptoms at all. Most people don’t. Your doctor could check you liver and see only a little damage. You might not get diagnosed until he spots a problem with your liver enzymes after a routine blood test.
Almost all people with hepatitis A recover fully in weeks or months. In contrast, hepatitis B and C can become chronic infections that lead to cirrhosis of the liver, liver cancer, and even death. What’s more, the three types differ in the way they spread from person to person:
Hepatitis A. The hepatitis A virus (HAV) is present in the feces of people who have hepatitis A and spreads by fecal-oral contact. Infection can occur if even a microscopic amount of virus-laden feces reaches the mouth. This can happen by consuming contaminated food or beverages, as well as through close personal contact or sex with an infected person.
Hepatitis B. The hepatitis B virus (HBV) is found in the blood, semen, vaginal secretions, and other body fluids of people who have hepatitis B. Infection occurs when there is contact with these fluids -- for example, during sex with an infected person or exposure to contaminated needles or personal items. Up to 25% of people with chronic HBV infection die from liver disease.
Hepatitis C. The hepatitis C virus (HCV) is found in the blood of people who have hepatitis C. It can be spread by sexual contact, although it’s usually spread from mother to child during childbirth or by sharing hypodermic needles or other drug paraphernalia. Up to 85% of people infected with HCV develop chronic hepatitis C infection.