Black lung disease is a common name for any lung disease that develops from inhaling coal dust. This name comes from the fact that those with the disease have lungs that look black instead of pink. Medically, it is a type of pneumoconiosis called coal workers' pneumoconiosis (CWP). There are two forms: simple CWP and complicated CWP, which also involves progressive massive fibrosis (PMF).
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The inhalation and accumulation of coal dust causes coal workers' pneumoconiosis (CWP). This stems from working in a coal mine, coal trimming (loading and stowing coal for storage), mining or milling graphite, and manufacturing carbon electrodes (used in certain types of large furnaces) and carbon black (a compound used in many items, such as tires and other rubber goods). Because CWP is a reaction to accumulated dust in the lungs, it may appear and get worse during your exposure to the dust or after your exposure has ceased.
The severity of CWP depends on the type of coal dust, how much dust was in the air, and how long you have been exposed to it.
Is CWP the same thing as chronic obstructive pulmonary disease (COPD)?
CWP starts with the inhalation and accumulation of coal dust in the lungs. For many, there are no symptoms or noticeable effect on quality of life. There may be a cough and sputum (mucus) from inhalation of coal dust, but this may be more a matter of dust-induced bronchitis. As CWP progresses and is complicated by PMF, a cough and shortness of breath develop, along with sputum and moderate to severe airway obstruction. Quality of life decreases. Complications of CWP include cor pulmonale.
Smoking does not increase the prevalence of CWP, nor does it affect the development of CWP. But it may add to lung damage and contribute to the development of COPD. Coal workers who smoke are at much greater risk of developing COPD than nonsmoking coal workers.